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Notably, the current literature remains asymmetric: kidney-to-lung mechanisms, particularly those involving circulating mtDAMPs and related inflammatory signaling, are supported by more concentrated evidence, whereas lung-to-kidney links are supported mainly by organ-level inflammatory, hypoxic, and hemodynamic mechanisms, with comparatively limited direct evidence linking pulmonary epithelial mitophagy to renal injury

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J Clin Endocrinol Metab 85 , 24022410 (2000)

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