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[2] Research into nootropics Scientific research on nootropics is ongoing, and so far, it presents a mixed picture
Under pathological conditions, FUNDC1 persists in a phosphorylated state that specifically impedes its molecular interaction with the autophagosomal marker protein LC3-II, consequently suppressing mitophagy initiation and ultimately inducing neuronal dysfunction ( Mitochondrial fragmentation has also been demonstrated in AD patients and model animals
Specifically, these alterations include an increased abundance of Proteobacteria and Lactobacillus , along with decreased levels of anti-inflammatory commensals such as Prevotella ( 58)
Herzig S, Shaw RJ
While current pharmacological treatments provide symptomatic relief and modestly slow disease progression, they fail to address the underlying pathophysiology and are often accompanied by severe adverse effects
