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The mechanism by which Semaglutide GLP-1 reduces belly fat is not fully understood, but it may be related to its effects on appetite and satiety, as well as its ability to improve insulin sensitivity and glucose uptake by muscle and fat tissues

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First, semaglutide activates GLP-1 receptors in the gastrointestinal tract, causing delayed gastric emptying

Nutrient-driven incretin secretion into intestinal lymph is different between diabetic Goto-Kakizaki rats and Wistar rats

Most patients experience mild to moderate nausea that tends to diminish over time as tolerance develops, typically improving after four to eight weeks of continued therapy